/* Sequence of slide descriptions */
desc11 = 'Case 1 - Figure 1
Low power view of eosinophilic esophagitis. The squamous mucosa is hyperplastic and there is an adherent inflammatory "pseudomembrane". ';
desc12 = 'Case 1 - Figure 2
Eosinophilic esophagitis. The squamous mucosa is infiltrated by numerous eosinophils with a predilection for the superficial half of the mucosa. The "pseudomembrane" is composed of numerous eosinophils within parakeratotic debris. ';
desc21 = 'Case 2 - Figure 1
A large pancreatic-type duct is surrounded by fibrous tissue in the muscular wall of the stomach. ';
desc22 = 'Case 2 - Figure 2
Lobular arrangements of mucous glands are also present between muscle bundles of the muscularis propria. ';
desc23 = 'Case 2 - Figure 3
Muscle tends to wrap around the glandular structures. ';
desc24 = 'Case 2 - Figure 4
Round nests of endocrine cells identical to normal pancreatic islets are scattered in the interstitium of the muscularis propria. ';
desc31 = 'Case 3 - Figure 1
Gross image of the subtotal gastrectomy specimen demonstrating thickened rugal folds and several polypoid nodules in the gastric body, ranging up to 1.3 cm in diameter. ';
desc32 = 'Case 3 - Figure 2
Low power photomicrograph of one of the polyps in the body mucosa, showing a carcinoid tumor, with invasion into the superficial submucosa. ';
desc33 = 'Case 3 - Figure 3
High power photomicrograph of the carcinoid tumor, here showing a pseudoacinar architecture and the uniform neoplastic cells characteristic of carcinoid tumors. ';
desc34 = 'Case 3 - Figure 4
Low power photomicrograph of the thickened body mucosa, which demonstrates marked hyperplasia and dilatation of the specialized oxyntic (fundic) glands. The overlying foveolar neck regions and surface epithelium are of normal thickness. ';
desc35 = 'Case 3 - Figure 5
In many areas of body mucosa, the parietal cells were cytologically abnormal, with vacuolated cytoplasm and cytoplasmic protrusions into dilated oxyntic glands. ';
desc36 = 'Case 3 - Figure 6
The carcinoid tumors arose in a background of nodular neuroendocrine (ECL cell) hyperplasia, which is evident here at the base of the gastric body mucosa. ';
desc41 = 'Case 4 - Figure 1
Low power view of section from gastric resection shows diffuse, extensive infiltration of the submucosa by amyloid. ';
desc42 = 'Case 4 - Figure 2
Higher power view shows extensive perivascular amyloid deposition with associated hemorrhage. ';
desc43a = 'Case 4 - Figure 3a
Amyloid appears as an amorphous, waxy, eosinophilic material on H&E staining (3A)... ';
desc43b = 'Case 4 - Figure 3b
...and upon Congo red staining has a red/green birefringent appearance under polarized light (3B). ';
desc51 = 'Case 5 - Figure 1
Most of the colon looked like this. There was prominent lymphoid hyperplasia at the mucosal-submucosal junction. Some follicles contained small granulomas, and some had small foci of mucosal inflammation including tiny ulcers, in other words, aphthous lesions. ';
desc52 = 'Case 5 - Figure 2
Superimposed on the features in the first slide, there were segments of transmural inflammation with ulcers, some of which were fissure ulcers. The inflammation obliterated the submucosa, extended through the muscularis propria, involved the pericolic adipose tissue and produced strictures. Loose granulomas were present from the submucosa through the pericolic adipose tissue.';
desc53 = 'Case 5 - Figure 3
In the submucosa of these strictures, there were numerous lymphocytic aggregates. ';
desc54 = 'Case 5 - Figure 4
In some places, numerous large loose granulomas almost filled the submucosa. ';
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