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Sunday, March 8, 2009 — 8:30 AM, Convention Center Korbell BRA
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- Severe Febrile Shock and Hemorrhage: Pathology, Pathogenesis, and Diverse Microbial Etiologies
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Moderators:
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David H. Walker, University of Texas Medical Branch, Galveston, TX and J. Stephen Dumler, the Johns Hopkins University School of Mediine, Baltimore, MD
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Disclosure:
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In accordance with ACCME guidelines regarding disclosure, the USCAP policy requires that faculty members who have a significant financial or other relationship with a commercial company, entity, or service (which will be discussed in this Symposium) must disclose this to attendees. The Academy also requires that speakers disclose any products that are not labeled for the use under discussion. The speakers listed below have indicated they have nothing to disclose.
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Click any blue title below to display and print the handout for individual lecture(s):
| 8:30 | Dengue Shock and Hemorrhagic Fever - Judith, Aronson, University of Texas Medical Branch, Galveston, TX |
| 9:15 | Malaria - Georges Grau, the University of Sydney, Sydney, Australia |
| 10:00 | Break |
| 10:30 | Bacterial Sepsis - Daniel Remick, Boston University School of Medicine and Boston Medical Center, Boston, MA |
| 11:15 | Yellow Fever - Sherif Zaki, Centers for Disease Control and Prevention, Atlanta, GA |
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Topics to be addressed were suggested and focus was established based on areas of perceived need for the enhanced education of all pathologists as determined by the expertise of the Program Committee members. While diagnosis and treatment of infectious diseases continues to be important, this session also introduces key concepts in understanding the pathologic and pathogenetic basis for the shock syndromes induced by selected pathogens. Speakers known to be highly competent pathologists who conduct primary research in the selected areas were chosen. The objective is to educate the attendees regarding how diverse etiologic agents and pathogenetic mechanisms can result in stereotypical pathophysiologic, pathological and histopathological responses among infected persons with shock and/or hemorrhage.
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